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Artigo de periodico
N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats (2016)
Thieme, Karina ; Silva, Karolline Santana da; Fabre, Nelly Takashima; Catazoni, Sergio; Monteiro, Maria Beatriz; Bezerra, Daniele Pereira Santos; Pessoa, Juliana Martins da Costa; Souza, Maria Oliveira de ; Machado, Ubiratan Fabres ; Passarelli, Marisa; Giannella, Maria Lucia Gardillo Correa
Source: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Subjects: ESTRESSE OXIDATIVO, NEFROPATIAS, RIM, RATOS
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ABNT
THIEME, Karina et al. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats. Cellular Physiology and Biochemistry, v. 40, n. 3-4, p. 608-620, 2016Tradução . . Disponível em: https://doi.org/10.1159/000452574. Acesso em: 03 maio 2024.
APA
Thieme, K., Silva, K. S. da, Fabre, N. T., Catazoni, S., Monteiro, M. B., Bezerra, D. P. S., et al. (2016). N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats. Cellular Physiology and Biochemistry, 40( 3-4), 608-620. doi:10.1159/000452574
NLM
Thieme K, Silva KS da, Fabre NT, Catazoni S, Monteiro MB, Bezerra DPS, Pessoa JM da C, Souza MO de, Machado UF, Passarelli M, Giannella MLGC. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats [Internet]. Cellular Physiology and Biochemistry. 2016 ; 40( 3-4): 608-620.[citado 2024 maio 03 ] Available from: https://doi.org/10.1159/000452574
Vancouver
Thieme K, Silva KS da, Fabre NT, Catazoni S, Monteiro MB, Bezerra DPS, Pessoa JM da C, Souza MO de, Machado UF, Passarelli M, Giannella MLGC. N-Acetyl Cysteine Attenuated the Deleterious Effects of Advanced Glycation End-Products on the Kidney of Non-Diabetic Rats [Internet]. Cellular Physiology and Biochemistry. 2016 ; 40( 3-4): 608-620.[citado 2024 maio 03 ] Available from: https://doi.org/10.1159/000452574
Artigo de periodico
A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) (2016)
Madeira, João L. O.; Jorge, Alexander Augusto de Lima; Martin, Regina M; Montenegro, Luciana Ribeiro; Franca, Marcela M.; Costalong, Everlayny F.; Correa, Fernanda A.; Otto, Aline P.; Arnhold, Ivo J. P; Freitas, Helayne S; Machado, Ubiratan Fabres ; Mendonça, Berenice Bilharinho de; Carvalho, Luciani R
Source: European journal of endocrinology. Unidades: FM, ICB
Subjects: GRAVIDEZ DE GÊMEOS, HORMÔNIO DO CRESCIMENTO, EXPRESSÃO GÊNICA
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ABNT
MADEIRA, João L. O. et al. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD). European journal of endocrinology, v. 175, n. 02, p. K7-K15, 2016Tradução . . Disponível em: https://doi.org/10.1530/eje-15-0149. Acesso em: 03 maio 2024.
APA
Madeira, J. L. O., Jorge, A. A. de L., Martin, R. M., Montenegro, L. R., Franca, M. M., Costalong, E. F., et al. (2016). A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD). European journal of endocrinology, 175( 02), K7-K15. doi:10.1530/eje-15-0149
NLM
Madeira JLO, Jorge AA de L, Martin RM, Montenegro LR, Franca MM, Costalong EF, Correa FA, Otto AP, Arnhold IJP, Freitas HS, Machado UF, Mendonça BB de, Carvalho LR. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) [Internet]. European journal of endocrinology. 2016 ; 175( 02): K7-K15.[citado 2024 maio 03 ] Available from: https://doi.org/10.1530/eje-15-0149
Vancouver
Madeira JLO, Jorge AA de L, Martin RM, Montenegro LR, Franca MM, Costalong EF, Correa FA, Otto AP, Arnhold IJP, Freitas HS, Machado UF, Mendonça BB de, Carvalho LR. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) [Internet]. European journal of endocrinology. 2016 ; 175( 02): K7-K15.[citado 2024 maio 03 ] Available from: https://doi.org/10.1530/eje-15-0149
Artigo de periodico
Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes (2016)
Monteiro, Maria Beatriz; Thieme, Karina ; Santos-Bezerra, Daniele Pereira; Queiroz, Márcia Silva; Woronik, Viktoria; Passarelli, Marisa; Machado, Ubiratan Fabres ; Giannella-Neto, Daniel; Oliveira-Souza, Maria; Corrêa-Giannella, Maria Lúcia
Source: Metabolism: Clinical and Experimental. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MONTEIRO, Maria Beatriz et al. Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes. Metabolism: Clinical and Experimental, v. 65, n. 6, p. 816-824, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2016.02.012. Acesso em: 03 maio 2024.
APA
Monteiro, M. B., Thieme, K., Santos-Bezerra, D. P., Queiroz, M. S., Woronik, V., Passarelli, M., et al. (2016). Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes. Metabolism: Clinical and Experimental, 65( 6), 816-824. doi:10.1016/j.metabol.2016.02.012
NLM
Monteiro MB, Thieme K, Santos-Bezerra DP, Queiroz MS, Woronik V, Passarelli M, Machado UF, Giannella-Neto D, Oliveira-Souza M, Corrêa-Giannella ML. Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes [Internet]. Metabolism: Clinical and Experimental. 2016 ; 65( 6): 816-824.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.metabol.2016.02.012
Vancouver
Monteiro MB, Thieme K, Santos-Bezerra DP, Queiroz MS, Woronik V, Passarelli M, Machado UF, Giannella-Neto D, Oliveira-Souza M, Corrêa-Giannella ML. Beta-2-microglobulin (B2M) expression in the urinary sediment correlates with clinical markers of kidney disease in patients with type 1 diabetes [Internet]. Metabolism: Clinical and Experimental. 2016 ; 65( 6): 816-824.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.metabol.2016.02.012
Artigo de periodico
Thioredoxin interacting protein expression in the urinary sediment associates with renal function decline in type 1 diabetes (2016)
Monteiro, Maria Beatriz; Santos-Bezerra, Daniele Pereira; Thieme, Karina ; Admoni, Sharon Nina; Perez, Ricardo Vessoni; Machado, Cleide Guimarães; Queiroz, Marcia Silva; Nery, Marcia; Oliveira-Souza, Maria; Woronik, Viktoria; Passarelli, Marisa; Giannella-Neto, Daniel; Machado, Ubiratan Fabres ; Corrêa-Giannella, M. Lúcia
Source: Free Radical Research. Unidades: FM, ICB
Subjects: DIABETES MELLITUS (COMPLICAÇÕES), HEPATOPATIAS, ESTRESSE OXIDATIVO, URINÁLISE
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ABNT
MONTEIRO, Maria Beatriz et al. Thioredoxin interacting protein expression in the urinary sediment associates with renal function decline in type 1 diabetes. Free Radical Research, v. 50, n. 1, p. 101-110, 2016Tradução . . Disponível em: http://www.tandfonline.com/doi/pdf/10.3109/10715762.2015.1109083. Acesso em: 03 maio 2024.
APA
Monteiro, M. B., Santos-Bezerra, D. P., Thieme, K., Admoni, S. N., Perez, R. V., Machado, C. G., et al. (2016). Thioredoxin interacting protein expression in the urinary sediment associates with renal function decline in type 1 diabetes. Free Radical Research, 50( 1), 101-110. doi:10.3109/10715762.2015.1109083
NLM
Monteiro MB, Santos-Bezerra DP, Thieme K, Admoni SN, Perez RV, Machado CG, Queiroz MS, Nery M, Oliveira-Souza M, Woronik V, Passarelli M, Giannella-Neto D, Machado UF, Corrêa-Giannella ML. Thioredoxin interacting protein expression in the urinary sediment associates with renal function decline in type 1 diabetes [Internet]. Free Radical Research. 2016 ; 50( 1): 101-110.[citado 2024 maio 03 ] Available from: http://www.tandfonline.com/doi/pdf/10.3109/10715762.2015.1109083
Vancouver
Monteiro MB, Santos-Bezerra DP, Thieme K, Admoni SN, Perez RV, Machado CG, Queiroz MS, Nery M, Oliveira-Souza M, Woronik V, Passarelli M, Giannella-Neto D, Machado UF, Corrêa-Giannella ML. Thioredoxin interacting protein expression in the urinary sediment associates with renal function decline in type 1 diabetes [Internet]. Free Radical Research. 2016 ; 50( 1): 101-110.[citado 2024 maio 03 ] Available from: http://www.tandfonline.com/doi/pdf/10.3109/10715762.2015.1109083
Artigo de periodico
In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux (2015)
Machado-Lima, Adriana; Iborra, Rodrigo T.; Pinto, Raphael S.; Castilho, Gabriela; Sartori, Camila H.; Oliveira, Erika R.; Okuda, Ligia S.; Nakandakare, Edna R.; Giannella-Neto, Daniel; Machado, Ubiratan Fabres ; Corrêa-Giannella, Maria Lucia C.; Traldi, Pietro; Porcu, Simona; Roverso, Marco; Lapolla, Annunziata; Passarelli, Marisa
Source: Journal of Cellular Physiology. Unidades: FM, ICB
Subjects: COLESTEROL, MACRÓFAGOS, DIABETES MELLITUS NÃO INSULINO-DEPENDENTE, EXPRESSÃO GÊNICA
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ABNT
MACHADO-LIMA, Adriana et al. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux. Journal of Cellular Physiology, v. 230, n. 6, p. 1250-1257, 2015Tradução . . Disponível em: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf. Acesso em: 03 maio 2024.
APA
Machado-Lima, A., Iborra, R. T., Pinto, R. S., Castilho, G., Sartori, C. H., Oliveira, E. R., et al. (2015). In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux. Journal of Cellular Physiology, 230( 6), 1250-1257. doi:10.1002/jcp.24860
NLM
Machado-Lima A, Iborra RT, Pinto RS, Castilho G, Sartori CH, Oliveira ER, Okuda LS, Nakandakare ER, Giannella-Neto D, Machado UF, Corrêa-Giannella MLC, Traldi P, Porcu S, Roverso M, Lapolla A, Passarelli M. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux [Internet]. Journal of Cellular Physiology. 2015 ; 230( 6): 1250-1257.[citado 2024 maio 03 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf
Vancouver
Machado-Lima A, Iborra RT, Pinto RS, Castilho G, Sartori CH, Oliveira ER, Okuda LS, Nakandakare ER, Giannella-Neto D, Machado UF, Corrêa-Giannella MLC, Traldi P, Porcu S, Roverso M, Lapolla A, Passarelli M. In Type 2 Diabetes Mellitus Glycated Albumin Alters Macrophage Gene Expression Impairing ABCA1-Mediated Cholesterol Efflux [Internet]. Journal of Cellular Physiology. 2015 ; 230( 6): 1250-1257.[citado 2024 maio 03 ] Available from: http://onlinelibrary.wiley.com/doi/10.1002/jcp.24860/epdf
Artigo de periodico
Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate (2015)
Christoffolete, Marcelo Augusto; Silva, William Jose; Ramos, Gracielle Vieira; Bento, Mirella Ribeiro; Costa, Monique Oliveira; Ribeiro, Miriam Oliveira; Okamoto, Maristela Mitiko; Tania Helena Lohmann; Machado, Ubiratan Fabres ; Musarò, Antonio; Moriscot, Anselmo Sigari
Source: Biomed Research International. Unidade: ICB
Subjects: ANATOMIA, BIOFÍSICA, FISIOLOGIA, SISTEMA MUSCULOSQUELÉTICO, INSULINA, CARBOIDRATOS, CAMUNDONGOS
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ABNT
CHRISTOFFOLETE, Marcelo Augusto et al. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate. Biomed Research International, v. 2015, p. 1-8, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/282984. Acesso em: 03 maio 2024.
APA
Christoffolete, M. A., Silva, W. J., Ramos, G. V., Bento, M. R., Costa, M. O., Ribeiro, M. O., et al. (2015). Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate. Biomed Research International, 2015, 1-8. doi:10.1155/2015/282984
NLM
Christoffolete MA, Silva WJ, Ramos GV, Bento MR, Costa MO, Ribeiro MO, Okamoto MM, Tania Helena Lohmann, Machado UF, Musarò A, Moriscot AS. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate [Internet]. Biomed Research International. 2015 ; 2015 1-8.[citado 2024 maio 03 ] Available from: https://doi.org/10.1155/2015/282984
Vancouver
Christoffolete MA, Silva WJ, Ramos GV, Bento MR, Costa MO, Ribeiro MO, Okamoto MM, Tania Helena Lohmann, Machado UF, Musarò A, Moriscot AS. Muscle IGF-1-induced skeletal muscle hypertrophy evokes higher insulin sensitivity and carbohydrate use as preferential energy substrate [Internet]. Biomed Research International. 2015 ; 2015 1-8.[citado 2024 maio 03 ] Available from: https://doi.org/10.1155/2015/282984
Artigo de periodico
Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats (2015)
Alves-Wagner, Ana Barbara; Mori, Rosana Cristina; Silva-Sabino, Robinson; Fatima, Luciana Alves; Alves, Adilson da Silva; Britto, Luiz Roberto Giorgetti de ; Schaan, Beatriz D'Agord; Machado, Ubiratan Fabres
Source: Autonomic Neuroscience: Basic and Clinical. Unidade: ICB
Subjects: INSULINA, RATOS WISTAR, GLICEMIA
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ABNT
ALVES-WAGNER, Ana Barbara et al. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats. Autonomic Neuroscience: Basic and Clinical, v. 193, p. 108-116, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2015.10.003. Acesso em: 03 maio 2024.
APA
Alves-Wagner, A. B., Mori, R. C., Silva-Sabino, R., Fatima, L. A., Alves, A. da S., Britto, L. R. G. de, et al. (2015). Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats. Autonomic Neuroscience: Basic and Clinical, 193, 108-116. doi:10.1016/j.autneu.2015.10.003
NLM
Alves-Wagner AB, Mori RC, Silva-Sabino R, Fatima LA, Alves A da S, Britto LRG de, Schaan BD'A, Machado UF. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2015 ; 193 108-116.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.autneu.2015.10.003
Vancouver
Alves-Wagner AB, Mori RC, Silva-Sabino R, Fatima LA, Alves A da S, Britto LRG de, Schaan BD'A, Machado UF. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2015 ; 193 108-116.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.autneu.2015.10.003
Artigo de periodico
Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells (2015)
Poletto, Ana Claudia; Furuya, Daniela T.; Silva, Aline David; Silva, Patricia E.; Santos, Camilo Lellis; Gianella, Maria Lucia C.; Passarelli, Marisa; Machado, Ubiratan Fabres
Source: Molecular and Cellular Endocrinology. Unidades: FM, ICB
Subjects: CÉLULAS MUSCULARES, ÁCIDOS GRAXOS NÃO SATURADOS, TÉCNICAS CITOLÓGICAS, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
POLETTO, Ana Claudia et al. Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells. Molecular and Cellular Endocrinology, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2014.12.001. Acesso em: 03 maio 2024.
APA
Poletto, A. C., Furuya, D. T., Silva, A. D., Silva, P. E., Santos, C. L., Gianella, M. L. C., et al. (2015). Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells. Molecular and Cellular Endocrinology. doi:10.1016/j.mce.2014.12.001
NLM
Poletto AC, Furuya DT, Silva AD, Silva PE, Santos CL, Gianella MLC, Passarelli M, Machado UF. Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells [Internet]. Molecular and Cellular Endocrinology. 2015 ;[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.mce.2014.12.001
Vancouver
Poletto AC, Furuya DT, Silva AD, Silva PE, Santos CL, Gianella MLC, Passarelli M, Machado UF. Oleic and linoleic fatty acids downregulate Slc2a4/GLUT4 expression via NFKB and SREBP1 in skeletal muscle cells [Internet]. Molecular and Cellular Endocrinology. 2015 ;[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.mce.2014.12.001
Artigo de periodico
Association of single nucleotide polymorphisms in the gene encoding GLUT1 and diabetic nephropathy in Brazilian patients with type 1 diabetes mellitus (2015)
Marques, T.; Patente, T. A.; Monteiro, M. B.; Cavaleiro, A. M.; Queiroz, M. S.; Nery, M.; Azevedo, M.J. de; Canani, L. H.; Parisi, M. C.; Moura-Neto, A.; Passarelli, M.; Gianella-Neto, D.; Machado, Ubiratan Fabres ; Corrêa-Giannella, M. L.
Source: Clinica Chimica Acta. Unidades: ICB, FM
Subjects: NEFROPATIAS, POLIMORFISMO, DIABETES MELLITUS (COMPLICAÇÕES)
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ABNT
MARQUES, T. et al. Association of single nucleotide polymorphisms in the gene encoding GLUT1 and diabetic nephropathy in Brazilian patients with type 1 diabetes mellitus. Clinica Chimica Acta, v. 444, p. 170-175, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.cca.2015.02.025. Acesso em: 03 maio 2024.
APA
Marques, T., Patente, T. A., Monteiro, M. B., Cavaleiro, A. M., Queiroz, M. S., Nery, M., et al. (2015). Association of single nucleotide polymorphisms in the gene encoding GLUT1 and diabetic nephropathy in Brazilian patients with type 1 diabetes mellitus. Clinica Chimica Acta, 444, 170-175. doi:10.1016/j.cca.2015.02.025
NLM
Marques T, Patente TA, Monteiro MB, Cavaleiro AM, Queiroz MS, Nery M, Azevedo MJ de, Canani LH, Parisi MC, Moura-Neto A, Passarelli M, Gianella-Neto D, Machado UF, Corrêa-Giannella ML. Association of single nucleotide polymorphisms in the gene encoding GLUT1 and diabetic nephropathy in Brazilian patients with type 1 diabetes mellitus [Internet]. Clinica Chimica Acta. 2015 ; 444 170-175.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.cca.2015.02.025
Vancouver
Marques T, Patente TA, Monteiro MB, Cavaleiro AM, Queiroz MS, Nery M, Azevedo MJ de, Canani LH, Parisi MC, Moura-Neto A, Passarelli M, Gianella-Neto D, Machado UF, Corrêa-Giannella ML. Association of single nucleotide polymorphisms in the gene encoding GLUT1 and diabetic nephropathy in Brazilian patients with type 1 diabetes mellitus [Internet]. Clinica Chimica Acta. 2015 ; 444 170-175.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.cca.2015.02.025
Artigo de periodico
Reduced Slc2a4/GLUT4 expression in subcutaneous adipose tissue of monosodium glutamate obese mice is recovered after atorvastatin treatment (2015)
Poletto, Ana Claudia; Silva, Aline David; Yamamoto, Aline Pedro de Melo; Machado, Ubiratan Fabres ; Furuya, Daniela Tomie
Source: Diabetology & Metabolic Syndrome. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, GLUTAMATOS, CAMUNDONGOS, HIPERCOLESTEROLEMIA
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ABNT
POLETTO, Ana Claudia et al. Reduced Slc2a4/GLUT4 expression in subcutaneous adipose tissue of monosodium glutamate obese mice is recovered after atorvastatin treatment. Diabetology & Metabolic Syndrome, v. 7, n. 18, p. 1-6, 2015Tradução . . Disponível em: https://doi.org/10.1186/s13098-015-0015-6. Acesso em: 03 maio 2024.
APA
Poletto, A. C., Silva, A. D., Yamamoto, A. P. de M., Machado, U. F., & Furuya, D. T. (2015). Reduced Slc2a4/GLUT4 expression in subcutaneous adipose tissue of monosodium glutamate obese mice is recovered after atorvastatin treatment. Diabetology & Metabolic Syndrome, 7( 18), 1-6. doi:10.1186/s13098-015-0015-6
NLM
Poletto AC, Silva AD, Yamamoto AP de M, Machado UF, Furuya DT. Reduced Slc2a4/GLUT4 expression in subcutaneous adipose tissue of monosodium glutamate obese mice is recovered after atorvastatin treatment [Internet]. Diabetology & Metabolic Syndrome. 2015 ; 7( 18): 1-6.[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/s13098-015-0015-6
Vancouver
Poletto AC, Silva AD, Yamamoto AP de M, Machado UF, Furuya DT. Reduced Slc2a4/GLUT4 expression in subcutaneous adipose tissue of monosodium glutamate obese mice is recovered after atorvastatin treatment [Internet]. Diabetology & Metabolic Syndrome. 2015 ; 7( 18): 1-6.[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/s13098-015-0015-6
Artigo de periodico
Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta (2015)
Pinto, Paula Ramos; Rocco, Débora Dias Ferraretto; Okuda, Ligia Shimabukuro; Machado-Lima, Adriana; Castilho, Gabriela; Silva, Karolline da Santana da; Gomes, Diego Juvenal; Pinto, Raphael de Souza; Iborra, Rodrigo Tallada; Ferreira, Guilherme da Silva; Nakandakare, Edna Regina; Machado, Ubiratan Fabres ; Correa-Giannella, Maria; Catanozi, Sergio; Passarelli, Marisa
Source: Lipids in Health and Disease. Unidades: FM, ICB
Subjects: EXPRESSÃO GÊNICA, MACRÓFAGOS, ARTERIOSCLEROSE (PREVENÇÃO E CONTROLE), EXERCÍCIO FÍSICO
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ABNT
PINTO, Paula Ramos et al. Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta. Lipids in Health and Disease, v. 14, p. 12 , 2015Tradução . . Disponível em: https://doi.org/10.1186/s12944-015-0093-3. Acesso em: 03 maio 2024.
APA
Pinto, P. R., Rocco, D. D. F., Okuda, L. S., Machado-Lima, A., Castilho, G., Silva, K. da S. da, et al. (2015). Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta. Lipids in Health and Disease, 14, 12 . doi:10.1186/s12944-015-0093-3
NLM
Pinto PR, Rocco DDF, Okuda LS, Machado-Lima A, Castilho G, Silva K da S da, Gomes DJ, Pinto R de S, Iborra RT, Ferreira G da S, Nakandakare ER, Machado UF, Correa-Giannella M, Catanozi S, Passarelli M. Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta [Internet]. Lipids in Health and Disease. 2015 ; 14 12 .[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/s12944-015-0093-3
Vancouver
Pinto PR, Rocco DDF, Okuda LS, Machado-Lima A, Castilho G, Silva K da S da, Gomes DJ, Pinto R de S, Iborra RT, Ferreira G da S, Nakandakare ER, Machado UF, Correa-Giannella M, Catanozi S, Passarelli M. Aerobic exercise training enhances the in vivo cholesterol trafficking from macrophages to the liver independently of changes in the expression of genes involved in lipid flux in macrophages and aorta [Internet]. Lipids in Health and Disease. 2015 ; 14 12 .[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/s12944-015-0093-3
Artigo de periodico
Glucose transporter 1 expression accompanies hypoxia sensing in the cyclic canine corpus luteum (2014)
Papa, Paula de Carvalho ; Sousa, Liza Margareth Medeiros de Carvalho; Silva, Renata dos Santos; Fonseca, Vanessa Uemura da ; Amaral, Vanessa Coutinho do; Hoffmann, Bernd; Alves-Wagner, Ana Bárbara; Machado, Ubiratan Fabres ; Kowalewski, Mariusz Pawel
Source: Reproduction. Unidades: FMVZ, ICB
Subjects: CÃES, REPRODUÇÃO ANIMAL, CORPO LÚTEO, CICLO ESTRAL ANIMAL, FIBROBLASTOS, ANÓXIA, ENDOTÉLIO VASCULAR
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ABNT
PAPA, Paula de Carvalho et al. Glucose transporter 1 expression accompanies hypoxia sensing in the cyclic canine corpus luteum. Reproduction, v. 147, n. 1, p. 81-889, 2014Tradução . . Disponível em: https://doi.org/10.1530/REP-13-0398. Acesso em: 03 maio 2024.
APA
Papa, P. de C., Sousa, L. M. M. de C., Silva, R. dos S., Fonseca, V. U. da, Amaral, V. C. do, Hoffmann, B., et al. (2014). Glucose transporter 1 expression accompanies hypoxia sensing in the cyclic canine corpus luteum. Reproduction, 147( 1), 81-889. doi:10.1530/REP-13-0398
NLM
Papa P de C, Sousa LMM de C, Silva R dos S, Fonseca VU da, Amaral VC do, Hoffmann B, Alves-Wagner AB, Machado UF, Kowalewski MP. Glucose transporter 1 expression accompanies hypoxia sensing in the cyclic canine corpus luteum [Internet]. Reproduction. 2014 ; 147( 1): 81-889.[citado 2024 maio 03 ] Available from: https://doi.org/10.1530/REP-13-0398
Vancouver
Papa P de C, Sousa LMM de C, Silva R dos S, Fonseca VU da, Amaral VC do, Hoffmann B, Alves-Wagner AB, Machado UF, Kowalewski MP. Glucose transporter 1 expression accompanies hypoxia sensing in the cyclic canine corpus luteum [Internet]. Reproduction. 2014 ; 147( 1): 81-889.[citado 2024 maio 03 ] Available from: https://doi.org/10.1530/REP-13-0398
Artigo de periodico
Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites (2014)
Moraes, Paulo Alexandre; Yonamine, Caio Yogi; Pinto Junior, Danilo Correa; Esteves, João Victor Del Conti; Machado, Ubiratan Fabres ; Mori, Rosana Cristina
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MORAES, Paulo Alexandre et al. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, v. 114, n. 1, p. 36-44, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.07.040. Acesso em: 03 maio 2024.
APA
Moraes, P. A., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., Machado, U. F., & Mori, R. C. (2014). Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, 114( 1), 36-44. doi:10.1016/j.lfs.2014.07.040
NLM
Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
Vancouver
Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
Artigo de periodico
Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes (2014)
Lehnen, Tatian Ederich; Lehnen, Alexandre Machado; Tavares, Angela Maria Vicente; Belló-Klein, Adriane; Markoski, Melissa Medeiros; Machado, Ubiratan Fabres ; Schaan, Beatriz
Source: Clinical and Experimental Pharmacology & Physiology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEHNEN, Tatian Ederich et al. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes. Clinical and Experimental Pharmacology & Physiology, v. 41, n. 12, p. 986-994, 2014Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12313. Acesso em: 03 maio 2024.
APA
Lehnen, T. E., Lehnen, A. M., Tavares, A. M. V., Belló-Klein, A., Markoski, M. M., Machado, U. F., & Schaan, B. (2014). Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes. Clinical and Experimental Pharmacology & Physiology, 41( 12), 986-994. doi:10.1111/1440-1681.12313
NLM
Lehnen TE, Lehnen AM, Tavares AMV, Belló-Klein A, Markoski MM, Machado UF, Schaan B. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes [Internet]. Clinical and Experimental Pharmacology & Physiology. 2014 ; 41( 12): 986-994.[citado 2024 maio 03 ] Available from: https://doi.org/10.1111/1440-1681.12313
Vancouver
Lehnen TE, Lehnen AM, Tavares AMV, Belló-Klein A, Markoski MM, Machado UF, Schaan B. Atorvastatin administered before myocardial infarction in rats improves contractility irrespective of metabolic changes [Internet]. Clinical and Experimental Pharmacology & Physiology. 2014 ; 41( 12): 986-994.[citado 2024 maio 03 ] Available from: https://doi.org/10.1111/1440-1681.12313
Artigo de periodico
Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis (2014)
Machado, Ubiratan Fabres ; Giannella, Maria Lúcia Cardillo Côrrea
Source: Expert Opinion on Emerging Drugs. Unidades: ICB, FM
Subjects: FISIOLOGIA, GLICOSE, DIABETES MELLITUS, HEMOGLOBINAS, MEDICAMENTO
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ABNT
MACHADO, Ubiratan Fabres e GIANNELLA, Maria Lúcia Cardillo Côrrea. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis. Expert Opinion on Emerging Drugs, v. 19, n. 1, p. 5-9, 2014Tradução . . Disponível em: https://doi.org/10.1517/14728214.2014.875530. Acesso em: 03 maio 2024.
APA
Machado, U. F., & Giannella, M. L. C. C. (2014). Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis. Expert Opinion on Emerging Drugs, 19( 1), 5-9. doi:10.1517/14728214.2014.875530
NLM
Machado UF, Giannella MLCC. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis [Internet]. Expert Opinion on Emerging Drugs. 2014 ; 19( 1): 5-9.[citado 2024 maio 03 ] Available from: https://doi.org/10.1517/14728214.2014.875530
Vancouver
Machado UF, Giannella MLCC. Sodium-glucose transporter 2 inhibitors in type 2 diabetes mellitus: navigating between Scylla and Charybdis [Internet]. Expert Opinion on Emerging Drugs. 2014 ; 19( 1): 5-9.[citado 2024 maio 03 ] Available from: https://doi.org/10.1517/14728214.2014.875530
Artigo de periodico
Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding (2014)
Zanquetta, Melissa Moreira; Alves-Wagner, Ana Barbara; Mori, Rosana Cristina; Campello, Raquel Saldanha; Machado, Ubiratan Fabres
Source: Metabolism: Clinical and Experimental. Unidade: ICB
Subjects: FISIOLOGIA, INSULINA, RNA, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZANQUETTA, Melissa Moreira et al. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding. Metabolism: Clinical and Experimental, v. 63, n. 3, p. 328-334, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2013.11.001. Acesso em: 03 maio 2024.
APA
Zanquetta, M. M., Alves-Wagner, A. B., Mori, R. C., Campello, R. S., & Machado, U. F. (2014). Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding. Metabolism: Clinical and Experimental, 63( 3), 328-334. doi:10.1016/j.metabol.2013.11.001
NLM
Zanquetta MM, Alves-Wagner AB, Mori RC, Campello RS, Machado UF. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding [Internet]. Metabolism: Clinical and Experimental. 2014 ; 63( 3): 328-334.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.metabol.2013.11.001
Vancouver
Zanquetta MM, Alves-Wagner AB, Mori RC, Campello RS, Machado UF. Recovery of insulin sensitivity and Slc2a4 mRNA expression depend on T3 hormone during refeeding [Internet]. Metabolism: Clinical and Experimental. 2014 ; 63( 3): 328-334.[citado 2024 maio 03 ] Available from: https://doi.org/10.1016/j.metabol.2013.11.001
Artigo de periodico
Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats (2014)
Yonamine, Caio Yogi; Teixeira, S. S.; Campello, R. S.; Gerlinger-Romero, Frederico; Rodrigues Jr, Carlos Flores; Guimarães-Ferreira, L.; Machado, Ubiratan Fabres ; Nunes, Maria Tereza
Source: Acta Physiologica. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YONAMINE, Caio Yogi et al. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats. Acta Physiologica, v. 212, n. 1, p. 62-74, 2014Tradução . . Disponível em: https://doi.org/10.1111/apha.12336. Acesso em: 03 maio 2024.
APA
Yonamine, C. Y., Teixeira, S. S., Campello, R. S., Gerlinger-Romero, F., Rodrigues Jr, C. F., Guimarães-Ferreira, L., et al. (2014). Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats. Acta Physiologica, 212( 1), 62-74. doi:10.1111/apha.12336
NLM
Yonamine CY, Teixeira SS, Campello RS, Gerlinger-Romero F, Rodrigues Jr CF, Guimarães-Ferreira L, Machado UF, Nunes MT. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats [Internet]. Acta Physiologica. 2014 ; 212( 1): 62-74.[citado 2024 maio 03 ] Available from: https://doi.org/10.1111/apha.12336
Vancouver
Yonamine CY, Teixeira SS, Campello RS, Gerlinger-Romero F, Rodrigues Jr CF, Guimarães-Ferreira L, Machado UF, Nunes MT. Beta hydroxy beta methylbutyrate supplementation impairs peripheral insulin sensitivity in healthy sedentary Wistar rats [Internet]. Acta Physiologica. 2014 ; 212( 1): 62-74.[citado 2024 maio 03 ] Available from: https://doi.org/10.1111/apha.12336
Artigo de periodico
Decreased diabetes-induced glycemic impairment in WKY and SHR involves enhanced skeletal muscle Slc2a4/GLUT4 expression (2014)
Alves-Wagner, Ana Barbara; Sabino-Silva, Robinson; Campello, Raquel S.; Mori, Rosana C.; Machado, Ubiratan Fabres
Source: Diabetology and Metabolic Syndrome. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ALVES-WAGNER, Ana Barbara et al. Decreased diabetes-induced glycemic impairment in WKY and SHR involves enhanced skeletal muscle Slc2a4/GLUT4 expression. Diabetology and Metabolic Syndrome, v. 6, n. 1, p. 1-5, 2014Tradução . . Disponível em: https://doi.org/10.1186/1758-5996-6-97. Acesso em: 03 maio 2024.
APA
Alves-Wagner, A. B., Sabino-Silva, R., Campello, R. S., Mori, R. C., & Machado, U. F. (2014). Decreased diabetes-induced glycemic impairment in WKY and SHR involves enhanced skeletal muscle Slc2a4/GLUT4 expression. Diabetology and Metabolic Syndrome, 6( 1), 1-5. doi:10.1186/1758-5996-6-97
NLM
Alves-Wagner AB, Sabino-Silva R, Campello RS, Mori RC, Machado UF. Decreased diabetes-induced glycemic impairment in WKY and SHR involves enhanced skeletal muscle Slc2a4/GLUT4 expression [Internet]. Diabetology and Metabolic Syndrome. 2014 ; 6( 1): 1-5.[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/1758-5996-6-97
Vancouver
Alves-Wagner AB, Sabino-Silva R, Campello RS, Mori RC, Machado UF. Decreased diabetes-induced glycemic impairment in WKY and SHR involves enhanced skeletal muscle Slc2a4/GLUT4 expression [Internet]. Diabetology and Metabolic Syndrome. 2014 ; 6( 1): 1-5.[citado 2024 maio 03 ] Available from: https://doi.org/10.1186/1758-5996-6-97
Artigo de periodico
High glucose concentration stimulates NHE-1 activity in distal nephron cells: the role of the Mek/Erk1/2/p90RSK and p38MAPK signaling pathways (2014)
Costa-Pessoa, Juliana Martins da; Damasceno, Rosélia Santos; Machado, Ubiratan Fabres ; Beloto-Silva, Olívia; Oliveira-Souza, Maria
Source: Celular Physiology and Biochemistry. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA-PESSOA, Juliana Martins da et al. High glucose concentration stimulates NHE-1 activity in distal nephron cells: the role of the Mek/Erk1/2/p90RSK and p38MAPK signaling pathways. Celular Physiology and Biochemistry, v. 33, n. 2, p. 333-343, 2014Tradução . . Disponível em: https://doi.org/10.1159/000356673. Acesso em: 03 maio 2024.
APA
Costa-Pessoa, J. M. da, Damasceno, R. S., Machado, U. F., Beloto-Silva, O., & Oliveira-Souza, M. (2014). High glucose concentration stimulates NHE-1 activity in distal nephron cells: the role of the Mek/Erk1/2/p90RSK and p38MAPK signaling pathways. Celular Physiology and Biochemistry, 33( 2), 333-343. doi:10.1159/000356673
NLM
Costa-Pessoa JM da, Damasceno RS, Machado UF, Beloto-Silva O, Oliveira-Souza M. High glucose concentration stimulates NHE-1 activity in distal nephron cells: the role of the Mek/Erk1/2/p90RSK and p38MAPK signaling pathways [Internet]. Celular Physiology and Biochemistry. 2014 ; 33( 2): 333-343.[citado 2024 maio 03 ] Available from: https://doi.org/10.1159/000356673
Vancouver
Costa-Pessoa JM da, Damasceno RS, Machado UF, Beloto-Silva O, Oliveira-Souza M. High glucose concentration stimulates NHE-1 activity in distal nephron cells: the role of the Mek/Erk1/2/p90RSK and p38MAPK signaling pathways [Internet]. Celular Physiology and Biochemistry. 2014 ; 33( 2): 333-343.[citado 2024 maio 03 ] Available from: https://doi.org/10.1159/000356673
Artigo de periodico
Changes in renal glucose transporters in an animal model of metabolic syndrome (2013)
Lehnen, A. M.; Leguisamo, N. M.; Dias, L. D.; Pinto, G. H.; Okamoto, N. M.; Machado, Ubiratan Fabres ; Schaan, B. D.
Source: Hormone and Metabolic Research. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LEHNEN, A. M. et al. Changes in renal glucose transporters in an animal model of metabolic syndrome. Hormone and Metabolic Research, v. 45, n. 11, p. 840-843 2013, 2013Tradução . . Disponível em: https://doi.org/10.1055/s-0033-1354381. Acesso em: 03 maio 2024.
APA
Lehnen, A. M., Leguisamo, N. M., Dias, L. D., Pinto, G. H., Okamoto, N. M., Machado, U. F., & Schaan, B. D. (2013). Changes in renal glucose transporters in an animal model of metabolic syndrome. Hormone and Metabolic Research, 45( 11), 840-843 2013. doi:10.1055/s-0033-1354381
NLM
Lehnen AM, Leguisamo NM, Dias LD, Pinto GH, Okamoto NM, Machado UF, Schaan BD. Changes in renal glucose transporters in an animal model of metabolic syndrome [Internet]. Hormone and Metabolic Research. 2013 ; 45( 11): 840-843 2013.[citado 2024 maio 03 ] Available from: https://doi.org/10.1055/s-0033-1354381
Vancouver
Lehnen AM, Leguisamo NM, Dias LD, Pinto GH, Okamoto NM, Machado UF, Schaan BD. Changes in renal glucose transporters in an animal model of metabolic syndrome [Internet]. Hormone and Metabolic Research. 2013 ; 45( 11): 840-843 2013.[citado 2024 maio 03 ] Available from: https://doi.org/10.1055/s-0033-1354381

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